Vitamin D Council podcast 08: Dr. JoEllen Welsh and breast cancer

Posted on: August 19, 2015   by  Vitamin D Council

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The eighth episode of the Vitamin D Council Podcast has arrived! You can listen to all previous episodes here.

We release one episode per month, each focused on a different area of research or theme related to vitamin D.

You can download and listen to the episodes in these blog posts on our website. All episodes are also available on  iTunes.

Here are the show notes for this episode:

In this episode, Dr. John Cannell interviews JoEllen Welsh, PhD. She is a world renowned expert in vitamin D and breast cancer research with 22 years of experience studying the basic mechanisms by which  vitamin D inhibits the growth of breast cancer.

Dr. Welsh is a professor at the University at Albany Cancer Research Center and has received several externally funded grants for her research efforts. Most recently, Dr. Welsh and her team received a $1.76 million 5-year grant from the National Institute in order to further their investigation on the mechanism behind vitamin D on triple negative breast cancer.

Dr. Welsh shares her expertise on vitamin D and breast cancer based on her extensive research efforts over the past several years. She also evaluates current epidemiological and in vitro studies and gives listeners a sneak peek into her current NIH funded research project.

Interviewed:

JoEllen Welsh, PhD.

Resources:

https://www.vitamindcouncil.org/tag/breast-cancer/

https://www.vitamindcouncil.org/health-conditions/breast-cancer/

 

 

 

 

2 Responses to Vitamin D Council podcast 08: Dr. JoEllen Welsh and breast cancer

  1. Rita Celone Umile

    This is amazing.

    (I know what I would do if I were diagnosed with any type of breast cancer).

    Thank you, Dr. Welsh, for your continued efforts in vitamin D research. I wish you much success.

  2. Ian

    Excellent. Thank you.
    How important is the retinoid X receptor in activating the VDR?
    Is there a relationship between RXR activation and the amount of exogenous vitamin D needed to “saturate” the VDR?
    Has anybody looked at the relationship between these in cancer cells?

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